infectedwith h2b mruby lentivirus (Addgene inc)
Addgene inc is a verified supplier 
93
Structured Review
Addgene inc
infectedwith h2b mruby lentivirus
Infectedwith H2b Mruby Lentivirus, supplied by Addgene inc, used in various techniques. Bioz Stars score: 93/100, based on 16 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/infectedwith+h2b+mruby+lentivirus/pLentiPGK+Hygro+DEST+H2B-mRuby2+(Plasmid+%2390236)/pm40885185-112-15-18
Average 93 stars, based on 16 article reviews
Infectedwith H2b Mruby Lentivirus, supplied by Addgene inc, used in various techniques. Bioz Stars score: 93/100, based on 16 PubMed citations. ZERO BIAS - scores, article reviews, protocol conditions and more
https://www.bioz.com/product/infectedwith+h2b+mruby+lentivirus/pLentiPGK+Hygro+DEST+H2B-mRuby2+(Plasmid+%2390236)/pm40885185-112-15-18
Average 93 stars, based on 16 article reviews
infectedwith h2b mruby lentivirus - by Bioz Stars,
2026-10
93/100 stars
Images
Related Articles
Isolation:Article Title: Pathogenic variants in TMEM184B cause a neurodevelopmental syndrome associated with alteration of metabolic signaling. Article Snippet: De novo TMEM184B variants in six children are associated with neurodevelopmental deficits hallmarked by developmental delay, corpus callosum hypoplasia, seizures, and/or microcephaly.. Studies in zebrafish, transformed cell lines, and induced pluripotent stem cells reveal disrupted neural development, apoptosis, and TFEB dysregulation, suggesting cellular metabolic disruption as a likely molecular mechanism. Flow Cytometry:Article Title: Pathogenic variants in TMEM184B cause a neurodevelopmental syndrome associated with alteration of metabolic signaling. Article Snippet: De novo TMEM184B variants in six children are associated with neurodevelopmental deficits hallmarked by developmental delay, corpus callosum hypoplasia, seizures, and/or microcephaly.. Studies in zebrafish, transformed cell lines, and induced pluripotent stem cells reveal disrupted neural development, apoptosis, and TFEB dysregulation, suggesting cellular metabolic disruption as a likely molecular mechanism. Plasmid Preparation:Article Title: Pathogenic variants in TMEM184B cause a neurodevelopmental syndrome associated with alteration of metabolic signaling. Article Snippet: De novo TMEM184B variants in six children are associated with neurodevelopmental deficits hallmarked by developmental delay, corpus callosum hypoplasia, seizures, and/or microcephaly.. Studies in zebrafish, transformed cell lines, and induced pluripotent stem cells reveal disrupted neural development, apoptosis, and TFEB dysregulation, suggesting cellular metabolic disruption as a likely molecular mechanism. |